Map of the short arm of chromosome 20. The red dot indicates the location of the PRNP gene.
At first, it was believed that the origin of CJD and other TSEs there Motorola is a "slow mobile phones virus (lentivirus) or other unknown. However, these have never been isolated. Furthermore, the agent that causes CJD has several features that are rare in microorganisms such as viruses and bacteria. Is immune to all common methods of sterilization, does not contain any genetic information in the form of nucleic acids (DNA or RNA) and usually a long incubation period before symptoms appear. In some cases, this period can be up to 40 years. The main scientific theory 'is now proven cellular phones to kuru, BSE, and cases of acquired CJD' claims that these TSEs are not caused by a microorganism but by a type cellular phone plans of protein called a prion.
Prions are in normal form cellular providers as a harmless protein found in cells of the body, which controls certain aspects of cellular life. However, the prion can also take a form capable of causing infectious disease. This is why the immune system is unable to combat prion, which is itself free phones a protein whose presence is normal in all cells of the body.
Harmless and infectious forms of prion protein and are almost identical, but the infectious form takes on a folded configuration different from the plans normal protein.
In acquired CJD, cell phones the prion enters the body through contact with infectious prions. In hereditary CJD, the wireless providers gene responsible for producing the normal protein has undergone a mutation that is only capable of producing the pathologic proteins.Perhaps the cause of the sporadic form is inexplicable that the normal prions become 'unknown reasons' in the version of the infectious wireless phones protein.
The most lethal of these Pathological prions is that even if one of them, this single molecule is able to "reconfigure" its similar to normal, producing a kind of unstoppable chain reaction that leaves the body without molecules "healthy."
Once listed, the abnormal prion proteins bind to form fibers or accumulations called "amyloid plaques" that can be seen under the microscope. Fibers and plaques may start to settle years cell phones before they start to show symptoms of Samsung CJD. It is still unclear the role of these structures in the disease or could affect the symptoms.
In hereditary TSEs have been identified several (up to 20) different mutations in the prion gene. The specific mutations that candy bar phone are found in each family may affect the type of TSE experiment, the frequency with which the disease appears in the family and the type of symptoms are more noticeable. However, not all people with mutations all the plans include in the gene acquiring CJD prions. This suggests that mutations may merely increase susceptibility to CJD and that perhaps there are still other unknown factors also LG play a role in the disease, without eliminating the infection through different routes.
See also: Prion
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